Search results for " Peripheral blood mononuclear cells"

showing 3 items of 3 documents

Expansion of intracellular IFN-γ positive lymphocytes during Mycoplasma agalactiae infection in sheep.

2010

Abstract A method to assess the expansion of antigen-specific intracellular IFN-γ positive T cell subsets during the infection will be helpful for a better understanding of mycoplasmal infections physiopathology in the sheep. We analysed the percentage of antigen-specific lymphocytes positive for intracellular IFN-γ during the infection of sheep with Mycoplasma agalactiae by culturing peripheral blood mononuclear cells of infected or uninfected animals with irradiated M. agalactiae . The expansion of antigen-specific IFN-γ positive lymphocytes in infected sheep was initially sustained by CD4 + T cells at day 15 after infection, when antigen specific IgG start to be detectable, followed by C…

Mycoplasma agalactiaeT cellved/biology.organism_classification_rank.speciesDouble negativeSheep DiseasesBiologyPeripheral blood mononuclear cellMycoplasma agalactiaeInterferon-gammaImmunityAntibody SpecificitymedicineCytotoxic T cellAnimalsMycoplasma InfectionsLymphocytesAgalactia Mastitis T cells Peripheral blood mononuclear cells CD4+ cells CD8+ cellsAntigens BacterialSheepGeneral Veterinaryved/biologyAntibodies Bacterialmedicine.anatomical_structureImmunoglobulin GImmunologyLeukocytes MononuclearIntracellularCD8Research in veterinary science
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Some Plant Defense Stimulators can induce IL-1β production in human immune cells in vitro

2020

8 pages; International audience; Among Plant Protection Products (PPP), a new emerging category of pesticides act by stimulating plant defense in order to improve plant resistance against microbial pathogens. Given that these compounds, the so-called Plant Defense Stimulators (PDS) act on innate immunity, we tested, using an in vitro approach on human mononuclear leucocytes (PBMC), the potential toxicity (XTT assay) and inflammatory effects (production of IL-1β) of 4 PPP belonging to different chemical families. We found that two products (LBG-01F34® and Regalis®) did not induce any cytotoxicity or IL-1 β production. The product BION-50 WG®, that contains Acibenzolar-S-methyl (ASM) and sili…

[SDV.IMM] Life Sciences [q-bio]/ImmunologyHealth Toxicology and Mutagenesismedicine.medical_treatmentXTT010501 environmental sciencesA vision for the future of pesticide toxicologyToxicology01 natural sciencesPeripheral blood mononuclear cellMicrobiology03 medical and health sciencesLaminarinchemistry.chemical_compound0302 clinical medicineImmune systemlcsh:RA1190-1270medicinePlant defense against herbivoryPesticidesCytotoxicitylcsh:Toxicology. Poisons0105 earth and related environmental sciencesInflammationCell metabolic activityInnate immune systemDanio rerioASM acyl-benzolar-S-methylZebra fishPBMCPlant Defense StimulatorIn vitro3. Good healthPPP Plant Protection productCytokinechemistryPDS Plant Defense StimulatorPBMC peripheral blood mononuclear cellsIL-1β[SDV.IMM]Life Sciences [q-bio]/Immunology030217 neurology & neurosurgeryToxicology Reports
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Apoptotic-like Leishmania exploit the host´s autophagy machinery to reduce T-cell-mediated parasite elimination

2015

Apoptosis is a well-defined cellular process in which a cell dies, characterized by cell shrinkage and DNA fragmentation. In parasites like Leishmania, the process of apoptosis-like cell death has been described. Moreover upon infection, the apoptotic-like population is essential for disease development, in part by silencing host phagocytes. Nevertheless, the exact mechanism of how apoptosis in unicellular organisms may support infectivity remains unclear. Therefore we investigated the fate of apoptotic-like Leishmania parasites in human host macrophages. Our data showed--in contrast to viable parasites--that apoptotic-like parasites enter an LC3(+), autophagy-like compartment. The compartm…

log.ph logarithmic phaseT-LymphocytesApoptosisMACS magnetic-associated cell sortingMacrophageMFI mean fluorescence intensityLeishmaniasisMOI multiplicity of infectionanti-inflammatoryLeishmaniaeducation.field_of_studyPhagocytesCFSE carboxyfluorescein succinimidyl esterTGFB transforming growth factorAcquired immune systemapoptotic-like LeishmaniaPS phosphatidylserinehuman primary macrophagesCell biologyβ; TT tetanus toxoidCorrigendumProgrammed cell deathautophagyPopulationAntigen presentationANXA5 annexin VBasic Science Research PapersBiologyPhagocytosisCM complete mediumMAP1LC3/LC3 microtubule-associated protein 1 light chain 3AnimalsHumansMHC major histocompatibility complexIF immunofluorescenceeducationMolecular Biologyimmune evasionPBMCs peripheral blood mononuclear cellsT-cell proliferationIntracellular parasiteMacrophagesstat.ph stationary phaseAutophagyLm LeishmaniaCell BiologyLeishmaniabiology.organism_classificationIL interleukinLAP LC3-associated phagocytosisLAPhMDM human monocyte derived macrophageAutophagy
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